Difference between revisions of "Changhan's Ph.D thesis"

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== Research interests and issues ==
 
== Research interests and issues ==
<br/> My interest is conquering aging.<br/> <br/> Aging is a progressive malfunctionization of information processor (e.g. our body). Degerative diseases are representative symptoms of aging.<br/> We can detect much evidence of aging through multiomics data. (genomics, transcriptome, epigenome, metabolome, etc.)<br/> <br/> Recently, researchers (such as David Sinclair) found some chemicals and genes that facilitate reverse aging.<br/> However,&nbsp;we could not yet find the mechanism of aging or reverse aging in detail.<br/> <br/> Aging simulation using multiomics data&nbsp;
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<br/> <span style="font-size:20px;">My interest is reverse aging.<br/> <br/> Aging is a progressive malfunctionization of information processor (e.g. our body). Degerative diseases are representative symptoms of aging.<br/> We can detect much evidence of aging through multiomics data. (genomics, transcriptome, epigenome, metabolome, etc.)<br/> <br/> Recently, researchers (such as David Sinclair) found some chemicals and genes that facilitate reverse aging.<br/> However,&nbsp;we could not yet find the mechanism of aging or reverse aging in detail.<br/> <br/> Aging is easily observable by eyes through appearances but the inner malfunctioning mechanisms is under cover.<br/> <br/> My hypothesis is that since aging occurs due to&nbsp;disturbed information propagations '''if we can&nbsp;find the method to resolve the propagation correctly, we can conquer aging.'''</span><br/> <br/> <img style="width: 996px; height: 518px;" src=http://Biolecture.org/upload/20230908152529_image.png>

Latest revision as of 15:31, 8 September 2023

Research interests and issues


My interest is reverse aging.

Aging is a progressive malfunctionization of information processor (e.g. our body). Degerative diseases are representative symptoms of aging.
We can detect much evidence of aging through multiomics data. (genomics, transcriptome, epigenome, metabolome, etc.)

Recently, researchers (such as David Sinclair) found some chemicals and genes that facilitate reverse aging.
However, we could not yet find the mechanism of aging or reverse aging in detail.

Aging is easily observable by eyes through appearances but the inner malfunctioning mechanisms is under cover.

My hypothesis is that since aging occurs due to disturbed information propagations if we can find the method to resolve the propagation correctly, we can conquer aging.